Publications
2007
Ferrandon Dominique, Gottar Marie, Gobert Vanessa
[New mechanism for detection of infections using the innate immune system of animals] Article de journal
Dans: Med Sci (Paris), vol. 23, no. 8-9, p. 707–709, 2007, ISSN: 0767-0974.
Liens | BibTeX | Étiquettes: Animal, Animals, Drosophila/immunology, ferrandon, Gram-Positive Bacteria, Gram-Positive Bacteria/pathogenicity, Gram-Positive Bacterial Infections, Gram-Positive Bacterial Infections/immunology, Humans, Immune System, infection, Infection/*diagnosis/*immunology, M3i, Models
@article{ferrandon_[new_2007b,
title = {[New mechanism for detection of infections using the innate immune system of animals]},
author = {Dominique Ferrandon and Marie Gottar and Vanessa Gobert},
doi = {10.1051/medsci/20072389707},
issn = {0767-0974},
year = {2007},
date = {2007-09-01},
journal = {Med Sci (Paris)},
volume = {23},
number = {8-9},
pages = {707--709},
keywords = {Animal, Animals, Drosophila/immunology, ferrandon, Gram-Positive Bacteria, Gram-Positive Bacteria/pathogenicity, Gram-Positive Bacterial Infections, Gram-Positive Bacterial Infections/immunology, Humans, Immune System, infection, Infection/*diagnosis/*immunology, M3i, Models},
pubstate = {published},
tppubtype = {article}
}
2004
Ferrandon Dominique, Imler Jean-Luc, Hoffmann Jules A
Sensing infection in Drosophila: Toll and beyond Article de journal
Dans: Semin Immunol, vol. 16, p. 43–53, 2004, ISSN: 1044-5323.
Résumé | BibTeX | Étiquettes: Animals, Carrier Proteins/chemistry/immunology/physiology, Cell Surface/immunology/*physiology, Drosophila Proteins/chemistry/immunology/*physiology, Drosophila/genetics/*immunology/microbiology, ferrandon, Fungi/immunology, Gene Expression Regulation, Gram-Negative Bacterial Infections/immunology, Gram-Positive Bacterial Infections/immunology, hoffmann, imler, Immunological, Insect Proteins/chemistry/immunology/physiology, M3i, Models, Non-U.S. Gov't, Receptors, Signal Transduction/immunology/physiology, Support
@article{ferrandon_sensing_2004b,
title = {Sensing infection in Drosophila: Toll and beyond},
author = {Dominique Ferrandon and Jean-Luc Imler and Jules A Hoffmann},
issn = {1044-5323},
year = {2004},
date = {2004-01-01},
journal = {Semin Immunol},
volume = {16},
pages = {43--53},
abstract = {Drosophila has evolved a potent immune system that is somewhat adapted to the nature of infections through the selective activation of either one of two NF-kappa B-like signalling pathways, the Toll and IMD (Immune deficiency) pathways. In contrast to the mammalian system, the Toll receptor does not act as a pattern recognition receptor (PRR) but as a cytokine receptor. The sensing of microbial infections is achieved by at least four PRRs that belong to two distinct families: the peptidoglycan recognition proteins (PGRPs) and the Gram-negative binding proteins (GNBPs)/beta-glucan recognition proteins (beta GRPs).},
keywords = {Animals, Carrier Proteins/chemistry/immunology/physiology, Cell Surface/immunology/*physiology, Drosophila Proteins/chemistry/immunology/*physiology, Drosophila/genetics/*immunology/microbiology, ferrandon, Fungi/immunology, Gene Expression Regulation, Gram-Negative Bacterial Infections/immunology, Gram-Positive Bacterial Infections/immunology, hoffmann, imler, Immunological, Insect Proteins/chemistry/immunology/physiology, M3i, Models, Non-U.S. Gov't, Receptors, Signal Transduction/immunology/physiology, Support},
pubstate = {published},
tppubtype = {article}
}